Quote:
Originally Posted by
uNiCoRnPriNcEsSx 
Try to get sum Bronkaid at your local pharmacy. 60 pills x 25 mg doses = 1.5 grams
Bronkaid contains guggulsterones, which has a myriad of problems with it. Chiefly that it inhibits the androgen receptors and "activates" the progesterone, which is why people get the hot flashes (though with all of the studies were done with high doses). Sure, Bronkaid works, but there are probably better and easier alternatives. On a related note, I found this while searching:
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Thus, both (E)- and (Z)-GS function as antagonists of MR, GR, and AR, and agonists of PR at concentrations well below those required to block FXR, indicating that the actions of GS may be caused by a polypharmacological profile of activity targeting the steroid receptor members of the nuclear hormone receptor superfamily in addition to FXR. Nuclear receptors such as FXR require significantly higher levels of their natural ligands (micromolar levels) for activation than do the steroid receptors (nanomolar levels).
However, based on our data indicating that GSs display considerably higher affinity for several steroid receptors than for FXR, it is reasonable to assume that doses of GS required to act on FXR in vivo would be well above those required to affect steroid receptor activity. Much of the activity that we have characterized for the GSs is consistent with the clinical pharmacology associated with its use.
It is clear that compounds with similar steroid receptor agonist/ antagonist profiles, such as cyproterone acetate, which functions as an AR and GR antagonist and PR agonist, have been demonstrated to have hypolipidemic activity (Damgaard- Pedersen and Fogh, 1980; Wallentin and Varenhorst, 1980). Likewise, spironolactone that functions as aMR and GR antagonist and PR agonist has been demonstrated to have beneficial effects on hypertension and congestive heart failure (Pitt et al., 1999). In addition, there are a variety of other pharmacological activities that have been associated with guggulipid use such as
reduction of acne (Urizar and Moore, 2003), which based on our data may be associated with the antiandrogen activity of the GSs. The range of receptors targeted by the GSs indicates that the pharmacology of these plant sterols will require greater examination to understand their activity and clinical significance.
http://molpharm.aspetjournals.org/content/67/3/948.full I read somewhere that guggulsterones were added to limit the affect of ephedrine on the CNS, but I can't find anything to substantiate that, so.... If the OP is in Canada he can buy Ephedrine HCL basically anywhere, in 8mg tabs in bottles of 50, with no quantity restriction or submission of ID to some regulatory body. For those of us in the land of the "free," Yohimbine HCL is a suitable replacement, though some people don't respond very well to it.
Quote:
Originally Posted by
D Yizz 
Tell me how are you planning to get ephedrine as a supplement in the US. It was stupidly banned a few years ago under some badly founded allegations..
Thank VPOTUS Biden for that.